Hormones are esterified to slow their release into the bloodstream and prolong their duration of action by increasing their fat solubility. The esters used are just carbon chains of varying shapes and sizes, with a carboxyl group bound to and cleaved from the alcohol on the D ring of the parent steroid. Generally speaking: the larger the carbon chain, the more nonpolar/hydrophobic the compound meaning the depot will stick around longer and the ester will be cleaved at a slower rate by the body's esterase enzymes. So testosterone propionate, having a three carbon ester, will be released bound and metabolized more quickly than testosterone enanthate which has a seven carbon chain attached. In addition to altering the rate of release, the ester in use will alter the relative amount of hormone per weight of total compound. IIRC test prop is ~84% test by weight while test e is ~72% which I think is where you're getting your 15% number. But I'm not solid on those numbers. Anyway, so those are the two big ways that the ester used alters the curve and AUC of hormone release. That's good that you used the same dose location because injection site does play a role in the release as well, and I'm not trying to downplay your effort. but precise consistency between blood samples would take some serious doing. What I think you're overlooking though is that while each ester has an accepted window for when to get your blood drawn, these times aren't perfect and still won't always give equivalent values as far as where along the curve of hormone release your value lies. That was convoluted but I can't think of how to explain it any better.
Oral Turinabol is suppressive to natural testosterone and should be used in conjunction with exogenous testosterone. Men who use Oral Turinabol without exogenous testosterone will risk a low testosterone condition. Such a condition can come with a host of possible symptoms ranging from physical, mental and sexually related. However, while physical related symptoms are unlikely when steroids are being used the others are a very real possibility.
Once the use of Oral Turinabol comes to an end natural testosterone production will begin again on its own. However, natural levels will still be very low and it will take a large amount of time to recover proper or healthy levels. For this reason most men are encouraged to implement a Post Cycle Therapy (PCT) plan once the use of anabolic steroids is discontinued. This will greatly speed up the recovery process and protect your lean tissue. Without a PCT plan it is possible for cortisol to become dominant for a period of time, destroy muscle tissue and promote fat gain. While a PCT plan will promote recovery, it will not return you to normal on its own. There is no PCT plan on earth that has this ability. However, a well planned PCT will speed up the process and ensure you have enough testosterone for proper bodily function while your levels continue to naturally rise.
There are a few important notes on natural recovery, the primary being that no low testosterone condition existed prior to anabolic steroid use. Further, natural recovery assumes no severe damage was done to the Hypothalamic-Pituitary-Testicular-Axis (HPTA) through improper anabolic steroid use. As a final note, women have no need to supplement with exogenous testosterone when using Oral Turinabol.
Regardless of your Dbol dosage, four to six weeks of use is about all anyone will ever need; some may extend to eight weeks total, but this should be rare and is normally not recommended. The reason for such short-term use once again revolves around this steroid's strong hepatotoxic nature, and if you ignore the recommended time frames you will put your liver at risk. Of course, as seen from the Dbol only cycle link, your total cycle should extend past this point of use or you will risk losing all your gains. A solid plan is an injectable or two with your Dianabol for six weeks, and then continuing the two injectable steroids for another twelve weeks.